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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
Fecha : |
23/03/2018 |
Actualizado : |
09/10/2019 |
Tipo de producción científica : |
Artículos en Revistas Indexadas Internacionales |
Autor : |
OLINDA, R. G.; MAIA, L. A.; FRADE, M. T. S.; SOARES, M. P.; BARROS, S. S.; DRIEMEIER, D.; RIET-CORREA, F.; DANTAS, A. F. M. |
Afiliación : |
Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG); Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG); Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG); Laboratório Regional de Diagnóstico, Faculdade de Veterinária (FV), Universidade Federal de Pelotas (UFPel); Laboratório Regional de Diagnóstico, Faculdade de Veterinária (FV), Universidade Federal de Pelotas (UFPel); Departamento de Patologia Clínica Veterinária, Faculdade de Veteriná- ria, Universidade Federal do Rio Grande do Sul (UFRGS),; FRANKLIN RIET-CORREA AMARAL, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG); Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG). |
Título : |
Degenerative axonopathy associated with copper deficiency in pigs. (Axonopatia degenerativa associada com deficiência de cobre em suínos). |
Fecha de publicación : |
2017 |
Fuente / Imprenta : |
Pesquisa Veterinária Brasileira, Rio de Janeiro v. 37, n. 9, p. 911-915, setembro. 2017. |
DOI : |
10.1590/S0100-736X2017000900002 |
Idioma : |
Inglés |
Notas : |
Article history: 1 Received on May 29, 2016 // Accepted for publication on November 11, 2016. |
Contenido : |
Abstract: The epidemiological, clinic and morphological (pathological and ultrastructural) aspects of four outbreaks of copper deficiency affecting 21- to 90-day-old pigs in the Northeast region of Brazil are reported. Clinical signs began with paraparesis and ataxia and progressed to flaccid or spastic paralysis of the pelvic and thoracic limbs, followed by sternal and/or lateral recumbence. In addition, some animals showed dog-sitting position and intention tremors. The clinical manifestation period was 5-20 days. Significant gross lesions were not observed; however, microscopically, symmetrical degeneration of the white matter with ballooned myelin sheaths containing occasional macrophages was observed, mainly in the spinal cord. Two pigs presented with necrosis ad loss of Purkinje cells and ectopic Purkinje cells in the granular layer and cerebellar white matter. A ultrastructural analysis showed different degrees of damage of myelinated axons in the spinal segments, including an absence of the axoplasm structures with only axonal residues remaining. The myelin sheaths were degenerated and often collapsed into the space previously occupied by the axon. These results suggest that myelin degeneration is secondary to the axonal lesion. Finally, the concentration of copper in the liver was determined using atomic absorption spectrophotometry and was found to be low (ranging from 2.2 to 10.8 ppm). In conclusion, in the Brazilian semiarid region, Cu deficiency occurs in 21 to 90-day-old pigs that ingested different types of waste in their food. MenosAbstract: The epidemiological, clinic and morphological (pathological and ultrastructural) aspects of four outbreaks of copper deficiency affecting 21- to 90-day-old pigs in the Northeast region of Brazil are reported. Clinical signs began with paraparesis and ataxia and progressed to flaccid or spastic paralysis of the pelvic and thoracic limbs, followed by sternal and/or lateral recumbence. In addition, some animals showed dog-sitting position and intention tremors. The clinical manifestation period was 5-20 days. Significant gross lesions were not observed; however, microscopically, symmetrical degeneration of the white matter with ballooned myelin sheaths containing occasional macrophages was observed, mainly in the spinal cord. Two pigs presented with necrosis ad loss of Purkinje cells and ectopic Purkinje cells in the granular layer and cerebellar white matter. A ultrastructural analysis showed different degrees of damage of myelinated axons in the spinal segments, including an absence of the axoplasm structures with only axonal residues remaining. The myelin sheaths were degenerated and often collapsed into the space previously occupied by the axon. These results suggest that myelin degeneration is secondary to the axonal lesion. Finally, the concentration of copper in the liver was determined using atomic absorption spectrophotometry and was found to be low (ranging from 2.2 to 10.8 ppm). In conclusion, in the Brazilian semiarid region, Cu deficiency occurs in 21 to ... Presentar Todo |
Palabras claves : |
Axonal degeneration; COPPER DEFICIENCY; DEFICIENCIA DE COBRE; Degeneração axonal; Degeneração da mielina; Degenerative myelopathy; Mielopatia; MINERAL DEFICIENCY; Myelin degeneration; PLATAFORMA SALUD ANIMAL; SWINE. |
Thesagro : |
DEFICIENCIAS DE MINERALES; ENFERMEDADES DE LOS ANIMALES; SUINOS. |
Asunto categoría : |
E16 Enfermedades de los animales |
URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/8985/1/Pesq.-Vet.-Bras.-379-911-915-setembro-2017-Olinda.pdf
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Marc : |
LEADER 02918naa a2200397 a 4500 001 1058315 005 2019-10-09 008 2017 bl uuuu u00u1 u #d 024 7 $a10.1590/S0100-736X2017000900002$2DOI 100 1 $aOLINDA, R. G. 245 $aDegenerative axonopathy associated with copper deficiency in pigs. (Axonopatia degenerativa associada com deficiência de cobre em suínos).$h[electronic resource] 260 $c2017 500 $aArticle history: 1 Received on May 29, 2016 // Accepted for publication on November 11, 2016. 520 $aAbstract: The epidemiological, clinic and morphological (pathological and ultrastructural) aspects of four outbreaks of copper deficiency affecting 21- to 90-day-old pigs in the Northeast region of Brazil are reported. Clinical signs began with paraparesis and ataxia and progressed to flaccid or spastic paralysis of the pelvic and thoracic limbs, followed by sternal and/or lateral recumbence. In addition, some animals showed dog-sitting position and intention tremors. The clinical manifestation period was 5-20 days. Significant gross lesions were not observed; however, microscopically, symmetrical degeneration of the white matter with ballooned myelin sheaths containing occasional macrophages was observed, mainly in the spinal cord. Two pigs presented with necrosis ad loss of Purkinje cells and ectopic Purkinje cells in the granular layer and cerebellar white matter. A ultrastructural analysis showed different degrees of damage of myelinated axons in the spinal segments, including an absence of the axoplasm structures with only axonal residues remaining. The myelin sheaths were degenerated and often collapsed into the space previously occupied by the axon. These results suggest that myelin degeneration is secondary to the axonal lesion. Finally, the concentration of copper in the liver was determined using atomic absorption spectrophotometry and was found to be low (ranging from 2.2 to 10.8 ppm). In conclusion, in the Brazilian semiarid region, Cu deficiency occurs in 21 to 90-day-old pigs that ingested different types of waste in their food. 650 $aDEFICIENCIAS DE MINERALES 650 $aENFERMEDADES DE LOS ANIMALES 650 $aSUINOS 653 $aAxonal degeneration 653 $aCOPPER DEFICIENCY 653 $aDEFICIENCIA DE COBRE 653 $aDegeneração axonal 653 $aDegeneração da mielina 653 $aDegenerative myelopathy 653 $aMielopatia 653 $aMINERAL DEFICIENCY 653 $aMyelin degeneration 653 $aPLATAFORMA SALUD ANIMAL 653 $aSWINE 700 1 $aMAIA, L. A. 700 1 $aFRADE, M. T. S. 700 1 $aSOARES, M. P. 700 1 $aBARROS, S. S. 700 1 $aDRIEMEIER, D. 700 1 $aRIET-CORREA, F. 700 1 $aDANTAS, A. F. M. 773 $tPesquisa Veterinária Brasileira, Rio de Janeiro$gv. 37, n. 9, p. 911-915, setembro. 2017.
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Registro original : |
INIA La Estanzuela (LE) |
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Biblioteca (s) : |
INIA Las Brujas. |
Fecha actual : |
01/04/2020 |
Actualizado : |
01/04/2020 |
Tipo de producción científica : |
Artículos en Revistas Indexadas Internacionales |
Circulación / Nivel : |
Internacional - -- |
Autor : |
HERNÁNDEZ-RODRÍGUEZ, L.; BERTALMIO, A.; RUBIO, L.; ROLON, R.; MAESO, D.; RIVAS, F. |
Afiliación : |
LESTER HERNÁNDEZ RODRÍGUEZ, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay.; ANA MARIA BERTALMIO CASARIEGO, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; LETICIA PAOLA RUBIO CATTANI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; RODOLFO ROQUE ROLON RODRIGUEZ, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; DIEGO CESAR MAESO TOZZI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; CARLOS FERNANDO RIVAS GRELA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
Título : |
Inability of the brown citrus aphid (Toxoptera citricida) to transmit citrus psorosis virus under controlled conditions. (Brief report). |
Fecha de publicación : |
2020 |
Fuente / Imprenta : |
Journal of Citrus Pathology, 2020, volume 7, issue 1, 45702. OPEN ACCESS. Permalink: https://escholarship.org/uc/item/8m62q3pj |
Idioma : |
Inglés |
Notas : |
Article history: Published online: 2020.
Supplementary material: https://escholarship.org/uc/item/8m62q3pj#supplemental
Corresponding author: L. Hernández-Rodríguez, email: lesterhernandeziift@gmail.com |
Contenido : |
ABSTRACT.
Might Toxoptera citricida (BrCA) be a citrus psorosis virus (CPsV) vector? We examined CPsV transmission by the BrCA throughout two experiments. In experiment 1, 4 CPsV-infected plants bearing BrCA colonies were introduced in separated cages with 12 healthy 'Madame Vinous' sweet orange (MV) seedlings in each one (48 in total). In experiment 2, 5 BrCAs collected from each CPsV-infected plant were transferred into 3 MV seedlings for each one (12 in total) and left for a 72-h inoculation period. Both experiments were replicated once. No psorosis symptoms or CPsV detection evidences a BrCA inability for CPsV transmission under our controlled conditions. |
Palabras claves : |
Aphid transmission; Aspiviridae; CPsV; Ophiovirus; Psorosis. |
Asunto categoría : |
H20 Enfermedades de las plantas |
URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/14350/1/qt8m62q3pj.pdf
https://escholarship.org/content/qt8m62q3pj/qt8m62q3pj.pdf?t=q47mtz
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Marc : |
LEADER 01679naa a2200253 a 4500 001 1060974 005 2020-04-01 008 2020 bl uuuu u00u1 u #d 100 1 $aHERNÁNDEZ-RODRÍGUEZ, L. 245 $aInability of the brown citrus aphid (Toxoptera citricida) to transmit citrus psorosis virus under controlled conditions. (Brief report).$h[electronic resource] 260 $c2020 500 $aArticle history: Published online: 2020. Supplementary material: https://escholarship.org/uc/item/8m62q3pj#supplemental Corresponding author: L. Hernández-Rodríguez, email: lesterhernandeziift@gmail.com 520 $aABSTRACT. Might Toxoptera citricida (BrCA) be a citrus psorosis virus (CPsV) vector? We examined CPsV transmission by the BrCA throughout two experiments. In experiment 1, 4 CPsV-infected plants bearing BrCA colonies were introduced in separated cages with 12 healthy 'Madame Vinous' sweet orange (MV) seedlings in each one (48 in total). In experiment 2, 5 BrCAs collected from each CPsV-infected plant were transferred into 3 MV seedlings for each one (12 in total) and left for a 72-h inoculation period. Both experiments were replicated once. No psorosis symptoms or CPsV detection evidences a BrCA inability for CPsV transmission under our controlled conditions. 653 $aAphid transmission 653 $aAspiviridae 653 $aCPsV 653 $aOphiovirus 653 $aPsorosis 700 1 $aBERTALMIO, A. 700 1 $aRUBIO, L. 700 1 $aROLON, R. 700 1 $aMAESO, D. 700 1 $aRIVAS, F. 773 $tJournal of Citrus Pathology, 2020, volume 7, issue 1, 45702. OPEN ACCESS. Permalink: https://escholarship.org/uc/item/8m62q3pj
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